Acquired nystagmus

Common Causes of Acquired Nystagmus

• Neurological disorders: Conditions such as stroke, multiple sclerosis, brain tumors, and Parkinson's disease can damage the parts of the brain responsible for eye movement control.

• Ear disorders: Inner ear problems, like vestibular neuritis or Meniere's disease, can disrupt the balance system and lead to nystagmus.

• Eye diseases: Conditions like cataracts, glaucoma, or retinal detachment can affect the visual system and trigger nystagmus.

• Medications: Certain medications can cause nystagmus as a side effect.

• Head injury: Trauma to the head can damage the brain and nerves involved in eye movement.

Anatomical Considerations

Acquired nystagmus can arise from damage to various components of the oculomotor system:

• Brainstem: The brainstem, which houses the nuclei for the cranial nerves controlling eye movement, can be affected by conditions like stroke or tumors.

• Cerebellum: The cerebellum, responsible for coordinating movements, can be damaged by disorders like multiple sclerosis or stroke.

• Inner ear: The vestibular system in the inner ear helps maintain balance and contributes to eye movement control. Damage to this system can lead to nystagmus.

• Extraocular muscles: Conditions like Graves' disease can cause inflammation of the extraocular muscles, leading to nystagmus.

Types of Acquired Nystagmus:

• Horizontal nystagmus: The most common type, where the eyes move horizontally back and forth.

• Vertical nystagmus: The eyes move up and down.

• Rotational nystagmus: The eyes move in a circular or torsional pattern.

Treatment: The treatment for acquired nystagmus depends on the underlying cause. It may involve medications, surgery, or rehabilitation therapy to address the underlying condition and improve eye movement control.

Note : This document serves educational purposes exclusively and does not constitute medical advice or treatment guidelines